Modulation of alternative splicing contributes to cancer development: focusing on p53 isoforms, p53β and p53γ
Cell Death Differ
.
2014 Sep;21(9):1347-9.
doi: 10.1038/cdd.2014.99.
Authors
H Solomon
1
,
M Sharon
2
,
V Rotter
1
Affiliations
1
Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot 76100, Israel.
2
Department of Biological Chemistry, Weizmann Institute of Science, Rehovot 76100, Israel.
PMID:
25109594
PMCID:
PMC4131187
DOI:
10.1038/cdd.2014.99
No abstract available
Publication types
Editorial
Research Support, Non-U.S. Gov't
Comment
MeSH terms
Alternative Splicing / genetics*
Humans
Tumor Suppressor Protein p53 / genetics*
Substances
TP53 protein, human
Tumor Suppressor Protein p53