Feng Y - Search Results

1

DNA adduct levels and intestinal lesions in congenic rapid and slow acetylator syrian hamsters admi food mutagens 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) or 2-amino-3-methylimidazo[4,5-f]quinoline (IQ).

Steffensen IL, Fretland AJ, Paulsen JE, Feng Y, Eide TJ, Devanaboyina US, Hein DW, Alexander J. Steffensen IL, et al. Among authors: feng y. Pharmacol Toxicol. 2000 Jun;86(6):257-63. doi: 10.1111/j.0901-9928.2000.860603.x. Pharmacol Toxicol. 2000. PMID: 10895988

2

Acetylator genotype-dependent formation of 2-aminofluorene-hemoglobin adducts in rapid and slow acetylator Syrian hamsters congenic at the NAT2 locus.

Feng Y, Rustan TD, Ferguson RJ, Doll MA, Hein DW. Feng Y, et al. Toxicol Appl Pharmacol. 1994 Jan;124(1):10-5. doi: 10.1006/taap.1994.1002. Toxicol Appl Pharmacol. 1994. PMID: 8291051

3

3,2'-Dimethyl-4-aminobiphenyl-DNA adduct formation in tumor target and nontarget organs of rapid and slow acetylator Syrian hamsters cogenic at the NAT2 locus.

Feng Y, Jiang W, Deitz AC, Hein DW. Feng Y, et al. Toxicol Appl Pharmacol. 1996 Oct;140(2):315-21. doi: 10.1006/taap.1996.0226. Toxicol Appl Pharmacol. 1996. PMID: 8887447

4

2-Aminofluorene-DNA adduct levels in tumor-target and nontarget organs of rapid and slow acetylator Syrian hamsters congenic at the NAT2 locus.

Feng Y, Jiang W, Hein DW. Feng Y, et al. Toxicol Appl Pharmacol. 1996 Nov;141(1):248-55. doi: 10.1006/taap.1996.0281. Toxicol Appl Pharmacol. 1996. PMID: 8917697

5

Cloning, sequencing, and recombinant expression of NAT1, NAT2, and NAT3 derived from the C3H/HeJ (rapid) and A/HeJ (slow) acetylator inbred mouse: functional characterization of the activation and deactivation of aromatic amine carcinogens.

Fretland AJ, Doll MA, Gray K, Feng Y, Hein DW. Fretland AJ, et al. Among authors: feng y. Toxicol Appl Pharmacol. 1997 Feb;142(2):360-6. doi: 10.1006/taap.1996.8036. Toxicol Appl Pharmacol. 1997. PMID: 9070359

6

Higher frequency of aberrant crypt foci in rapid than slow acetylator inbred rats administered the colon carcinogen 3,2'-dimethyl-4-aminobiphenyl.

Feng Y, Fretland AJ, Rustan TD, Jiang W, Becker WK, Hein DW. Feng Y, et al. Toxicol Appl Pharmacol. 1997 Nov;147(1):56-62. doi: 10.1006/taap.1997.8259. Toxicol Appl Pharmacol. 1997. PMID: 9356307

7

Higher DNA adduct levels in urinary bladder and prostate of slow acetylator inbred rats administered 3,2'-dimethyl-4-aminobiphenyl.

Jiang W, Feng Y, Hein DW. Jiang W, et al. Among authors: feng y. Toxicol Appl Pharmacol. 1999 May 1;156(3):187-94. doi: 10.1006/taap.1998.8621. Toxicol Appl Pharmacol. 1999. PMID: 10222311

8

Oral administration of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) yields PhIP-DNA adducts but not tumors in male Syrian hamsters congenic at the N-acetyltransferase 2 (NAT2) locus.

Fretland AJ, Devanaboyina US, Feng Y, Leff MA, Xiao GH, Webb SJ, Hein DW. Fretland AJ, et al. Among authors: feng y. Toxicol Sci. 2001 Feb;59(2):226-30. doi: 10.1093/toxsci/59.2.226. Toxicol Sci. 2001. PMID: 11158715

9

Construction of Syrian hamster lines congenic at the polymorphic acetyltransferase locus (NAT2): acetylator genotype-dependent N- and O-acetylation of arylamine carcinogens.

Hein DW, Doll MA, Rustan TD, Gray K, Ferguson RJ, Feng Y. Hein DW, et al. Among authors: feng y. Toxicol Appl Pharmacol. 1994 Jan;124(1):16-24. doi: 10.1006/taap.1994.1003. Toxicol Appl Pharmacol. 1994. PMID: 8291057

10

Prostate expression of N-acetyltransferase 1 (NAT1) and 2 (NAT2) in rapid and slow acetylator congenic Syrian hamster.

Hein DW, Doll MA, Xiao GH, Feng Y. Hein DW, et al. Among authors: feng y. Pharmacogenetics. 2003 Mar;13(3):159-67. doi: 10.1097/00008571-200303000-00005. Pharmacogenetics. 2003. PMID: 12618593

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