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[Esophageal lesions associated with diphosphonates].
Larsen KO, Stray N, Engh V, Sandnes D. Larsen KO, et al. Among authors: sandnes d. Tidsskr Nor Laegeforen. 2000 Aug 30;120(20):2397-9. Tidsskr Nor Laegeforen. 2000. PMID: 11475224 Free article. Norwegian.
Response to transforming growth factor alpha (TGFalpha) and epidermal growth factor (EGF) in hepatocytes: lower EGF receptor affinity of TGFalpha is associated with more sustained activation of p42/p44 mitogen-activated protein kinase and greater efficacy in stimulation of DNA synthesis.
Thoresen GH, Guren TK, Sandnes D, Peak M, Agius L, Christoffersen T. Thoresen GH, et al. Among authors: sandnes d. J Cell Physiol. 1998 Apr;175(1):10-8. doi: 10.1002/(SICI)1097-4652(199804)175:1<10::AID-JCP2>3.0.CO;2-F. J Cell Physiol. 1998. PMID: 9491776
Role of diacylglycerol (DAG) in hormonal induction of S phase in hepatocytes: the DAG-dependent protein kinase C pathway is not activated by epidermal growth factor (EGF), but is involved in mediating the enhancement of responsiveness to EGF by vasopressin, angiotensin II, and norepinephrine.
Dajani OF, Sandnes D, Melien O, Rezvani F, Nilssen LS, Thoresen GH, Christoffersen T. Dajani OF, et al. Among authors: sandnes d. J Cell Physiol. 1999 Aug;180(2):203-14. doi: 10.1002/(SICI)1097-4652(199908)180:2<203::AID-JCP8>3.0.CO;2-T. J Cell Physiol. 1999. PMID: 10395290
Activation of p42/p44 mitogen-activated protein kinase by angiotensin II, vasopressin, norepinephrine, and prostaglandin F2alpha in hepatocytes is sustained, and like the effect of epidermal growth factor, mediated through pertussis toxin-sensitive mechanisms.
Melien O, Thoresen GH, Sandnes D, Ostby E, Christoffersen T. Melien O, et al. Among authors: sandnes d. J Cell Physiol. 1998 Jun;175(3):348-58. doi: 10.1002/(SICI)1097-4652(199806)175:3<348::AID-JCP13>3.0.CO;2-1. J Cell Physiol. 1998. PMID: 9572480
57 results