Rönnstrand L - Search Results

1

Chk1 regulates the S phase checkpoint by coupling the physiological turnover and ionizing radiation-induced accelerated proteolysis of Cdc25A.

Sørensen CS, Syljuåsen RG, Falck J, Schroeder T, Rönnstrand L, Khanna KK, Zhou BB, Bartek J, Lukas J. Sørensen CS, et al. Among authors: ronnstrand l. Cancer Cell. 2003 Mar;3(3):247-58. doi: 10.1016/s1535-6108(03)00048-5. Cancer Cell. 2003. PMID: 12676583 Free article.

2

Distinct versus redundant properties among members of the INK4 family of cyclin-dependent kinase inhibitors.

Thullberg M, Bartkova J, Khan S, Hansen K, Rönnstrand L, Lukas J, Strauss M, Bartek J. Thullberg M, et al. Among authors: ronnstrand l. FEBS Lett. 2000 Mar 24;470(2):161-6. doi: 10.1016/s0014-5793(00)01307-7. FEBS Lett. 2000. PMID: 10734227 Free article.

3

Phosphorylation-dependent and -independent functions of p130 cooperate to evoke a sustained G1 block.

Hansen K, Farkas T, Lukas J, Holm K, Rönnstrand L, Bartek J. Hansen K, et al. Among authors: ronnstrand l. EMBO J. 2001 Feb 1;20(3):422-32. doi: 10.1093/emboj/20.3.422. EMBO J. 2001. PMID: 11157749 Free PMC article.

4

TNF-alpha suppresses the PDGF beta-receptor kinase.

Molander C, Kallin A, Izumi H, Rönnstrand L, Funa K. Molander C, et al. Among authors: ronnstrand l. Exp Cell Res. 2000 Jul 10;258(1):65-71. doi: 10.1006/excr.2000.4917. Exp Cell Res. 2000. PMID: 10912788

5

Mechanisms of platelet-derived growth factor-induced chemotaxis.

Rönnstrand L, Heldin CH. Rönnstrand L, et al. Int J Cancer. 2001 Mar 15;91(6):757-62. doi: 10.1002/1097-0215(200002)9999:9999<::aid-ijc1136>3.0.co;2-j. Int J Cancer. 2001. PMID: 11275976 Free article. Review. No abstract available.

6

Selective platelet-derived growth factor receptor kinase blockers reverse sis-transformation.

Kovalenko M, Gazit A, Böhmer A, Rorsman C, Rönnstrand L, Heldin CH, Waltenberger J, Böhmer FD, Levitzki A. Kovalenko M, et al. Among authors: ronnstrand l. Cancer Res. 1994 Dec 1;54(23):6106-14. Cancer Res. 1994. PMID: 7954456

7

Phosphorylation of a 72-kDa protein in PDGF-stimulated cells which forms complex with c-Crk, c-Fyn and Eps15.

Hansen K, Rönnstrand L, Claesson-Welsh L, Heldin CH. Hansen K, et al. Among authors: ronnstrand l. FEBS Lett. 1997 Jun 9;409(2):195-200. doi: 10.1016/s0014-5793(97)00495-x. FEBS Lett. 1997. PMID: 9202144 Free article.

8

PDGF-induced phosphorylation of Tyr28 in the N-terminus of Fyn affects Fyn activation.

Hansen K, Alonso G, Courtneidge SA, Rönnstrand L, Heldin CH. Hansen K, et al. Among authors: ronnstrand l. Biochem Biophys Res Commun. 1997 Dec 18;241(2):355-62. doi: 10.1006/bbrc.1997.7743. Biochem Biophys Res Commun. 1997. PMID: 9425276

9

Mutation of a Src phosphorylation site in the PDGF beta-receptor leads to increased PDGF-stimulated chemotaxis but decreased mitogenesis.

Hansen K, Johnell M, Siegbahn A, Rorsman C, Engström U, Wernstedt C, Heldin CH, Rönnstrand L. Hansen K, et al. Among authors: ronnstrand l. EMBO J. 1996 Oct 1;15(19):5299-313. EMBO J. 1996. PMID: 8895575 Free PMC article.

10

Overactivation of phospholipase C-gamma1 renders platelet-derived growth factor beta-receptor-expressing cells independent of the phosphatidylinositol 3-kinase pathway for chemotaxis.

Rönnstrand L, Siegbahn A, Rorsman C, Johnell M, Hansen K, Heldin CH. Rönnstrand L, et al. J Biol Chem. 1999 Jul 30;274(31):22089-94. doi: 10.1074/jbc.274.31.22089. J Biol Chem. 1999. PMID: 10419537 Free article.

. , Rorsman, C., Engstrom, U., Wernstedt, C., Heldin, C.-H., and Ronnstrand, L. (1996) EMBO J. 15, 5299-5313). Here we show that the increased chemotaxis correlates with increased activation of phospholipase C-gamma1 (PLC-gamma1), measured as inositol-1,4, 5-trispho …

. , Rorsman, C., Engstrom, U., Wernstedt, C., Heldin, C.-H., and Ronnstrand, L. (1996) EMBO J. 15, 5299-5313). Here we show th …

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