Scharnagl H - Search Results

1

The apolipoprotein E polymorphism is associated with circulating C-reactive protein (the Ludwigshafen risk and cardiovascular health study).

März W, Scharnagl H, Hoffmann MM, Boehm BO, Winkelmann BR. März W, et al. Among authors: scharnagl h. Eur Heart J. 2004 Dec;25(23):2109-19. doi: 10.1016/j.ehj.2004.08.024. Eur Heart J. 2004. PMID: 15571826

2

Apolipoprotein E polymorphism influences not only cerebral senile plaque load but also Alzheimer-type neurofibrillary tangle formation.

Ohm TG, Kirca M, Bohl J, Scharnagl H, Gross W, März W. Ohm TG, et al. Among authors: scharnagl h. Neuroscience. 1995 Jun;66(3):583-7. doi: 10.1016/0306-4522(94)00596-w. Neuroscience. 1995. PMID: 7644022

3

A novel assay for cytosolic 3-hydroxy-3-methylglutaryl-coenzyme A synthase activity using reversed-phase ion-pair chromatography: demonstration that Lifibrol (K12.148) modulates the enzyme activity.

Scharnagl H, März W, Schliack M, Löser R, Gross W. Scharnagl H, et al. J Lipid Res. 1995 Mar;36(3):622-7. J Lipid Res. 1995. PMID: 7775873 Free article.

4

Decreased binding of apolipoprotein (a) to familial defective apolipoprotein B-100 (Arg3500-->Gln). A study of the assembly of recombinant apolipoprotein (a) with mutant low density lipoproteins.

Durovic S, März W, Frank S, Scharnagl H, Baumstark MW, Zechner R, Kostner GM. Durovic S, et al. Among authors: scharnagl h. J Biol Chem. 1994 Dec 2;269(48):30320-5. J Biol Chem. 1994. PMID: 7982944 Free article.

5

Accumulation of "small dense" low density lipoproteins (LDL) in a homozygous patients with familial defective apolipoprotein B-100 results from heterogenous interaction of LDL subfractions with the LDL receptor.

März W, Baumstark MW, Scharnagl H, Ruzicka V, Buxbaum S, Herwig J, Pohl T, Russ A, Schaaf L, Berg A, et al. März W, et al. Among authors: scharnagl h. J Clin Invest. 1993 Dec;92(6):2922-33. doi: 10.1172/JCI116915. J Clin Invest. 1993. PMID: 8254047 Free PMC article.

6

Apolipoprotein E polymorphism is associated with both senile plaque load and Alzheimer-type neurofibrillary tangle formation.

März W, Scharnagl H, Kirça M, Bohl J, Gross W, Ohm TG. März W, et al. Among authors: scharnagl h. Ann N Y Acad Sci. 1996 Jan 17;777:276-80. doi: 10.1111/j.1749-6632.1996.tb34432.x. Ann N Y Acad Sci. 1996. PMID: 8624098

7

The receptor-mediated endocytosis of lipoprotein (a).

März W, Scharnagl H, Mondorf U, Wieland H, Gross W, Kostner GM. März W, et al. Among authors: scharnagl h. Z Gastroenterol. 1996 Jun;34 Suppl 3:131-8. Z Gastroenterol. 1996. PMID: 8767490 No abstract available.

8

Apolipoprotein E5 (Glu212-->Lys): increased binding to cell surface proteoglycans but decreased uptake and lysosomal degradation in cultured fibroblasts.

Feussner G, Scharnagl H, Scherbaum C, Acar J, Dobmeyer J, Lohrmann J, Wieland H, März W. Feussner G, et al. Among authors: scharnagl h. J Lipid Res. 1996 Aug;37(8):1632-45. J Lipid Res. 1996. PMID: 8864947 Free article.

9

Homozygous familial defective apolipoprotein B-100. Enhanced removal of apolipoprotein E-containing VLDLs and decreased production of LDLs.

Schaefer JR, Scharnagl H, Baumstark MW, Schweer H, Zech LA, Seyberth H, Winkler K, Steinmetz A, März W. Schaefer JR, et al. Among authors: scharnagl h. Arterioscler Thromb Vasc Biol. 1997 Feb;17(2):348-53. doi: 10.1161/01.atv.17.2.348. Arterioscler Thromb Vasc Biol. 1997. PMID: 9081691

10

Apolipoprotein E2 (Arg136 --> Cys) mutation in the receptor binding domain of apoE is not associated with dominant type III hyperlipoproteinemia.

März W, Hoffmann MM, Scharnagl H, Fisher E, Chen M, Nauck M, Feussner G, Wieland H. März W, et al. Among authors: scharnagl h. J Lipid Res. 1998 Mar;39(3):658-69. J Lipid Res. 1998. PMID: 9548597 Free article.

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