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Non-fibrillar beta-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptors.
Shemer I, Holmgren C, Min R, Fülöp L, Zilberter M, Sousa KM, Farkas T, Härtig W, Penke B, Burnashev N, Tanila H, Zilberter Y, Harkany T. Shemer I, et al. Among authors: penke b. Eur J Neurosci. 2006 Apr;23(8):2035-47. doi: 10.1111/j.1460-9568.2006.04733.x. Eur J Neurosci. 2006. PMID: 16630051
Amyloid beta-induced neuronal hyperexcitability triggers progressive epilepsy.
Minkeviciene R, Rheims S, Dobszay MB, Zilberter M, Hartikainen J, Fülöp L, Penke B, Zilberter Y, Harkany T, Pitkänen A, Tanila H. Minkeviciene R, et al. Among authors: penke b. J Neurosci. 2009 Mar 18;29(11):3453-62. doi: 10.1523/JNEUROSCI.5215-08.2009. J Neurosci. 2009. PMID: 19295151 Free PMC article.
Acute intracerebral treatment with amyloid-beta (1-42) alters the profile of neuronal oscillations that accompany LTP induction and results in impaired LTP in freely behaving rats.
Kalweit AN, Yang H, Colitti-Klausnitzer J, Fülöp L, Bozsó Z, Penke B, Manahan-Vaughan D. Kalweit AN, et al. Among authors: penke b. Front Behav Neurosci. 2015 May 6;9:103. doi: 10.3389/fnbeh.2015.00103. eCollection 2015. Front Behav Neurosci. 2015. PMID: 25999827 Free PMC article.
292 results