Mirabella M - Search Results

1

The recovery of platelet cyclooxygenase activity explains interindividual variability in responsiveness to low-dose aspirin in patients with and without diabetes.

Rocca B, Santilli F, Pitocco D, Mucci L, Petrucci G, Vitacolonna E, Lattanzio S, Mattoscio D, Zaccardi F, Liani R, Vazzana N, Del Ponte A, Ferrante E, Martini F, Cardillo C, Morosetti R, Mirabella M, Ghirlanda G, Davì G, Patrono C. Rocca B, et al. Among authors: mirabella m. J Thromb Haemost. 2012 Jul;10(7):1220-30. doi: 10.1111/j.1538-7836.2012.04723.x. J Thromb Haemost. 2012. PMID: 22471290 Free article. Clinical Trial.

2

The PPARgamma ligands PGJ2 and rosiglitazone show a differential ability to inhibit proliferation and to induce apoptosis and differentiation of human glioblastoma cell lines.

Morosetti R, Servidei T, Mirabella M, Rutella S, Mangiola A, Maira G, Mastrangelo R, Koeffler HP. Morosetti R, et al. Among authors: mirabella m. Int J Oncol. 2004 Aug;25(2):493-502. Int J Oncol. 2004. PMID: 15254749

3

alpha-Dystroglycan does not play a major pathogenic role in autosomal recessive hereditary inclusion-body myopathy.

Broccolini A, Gliubizzi C, Pavoni E, Gidaro T, Morosetti R, Sciandra F, Giardina B, Tonali P, Ricci E, Brancaccio A, Mirabella M. Broccolini A, et al. Among authors: mirabella m. Neuromuscul Disord. 2005 Feb;15(2):177-84. doi: 10.1016/j.nmd.2004.10.001. Epub 2004 Dec 10. Neuromuscul Disord. 2005. PMID: 15694140

4

Neprilysin participates in skeletal muscle regeneration and is accumulated in abnormal muscle fibres of inclusion body myositis.

Broccolini A, Gidaro T, Morosetti R, Gliubizzi C, Servidei T, Pescatori M, Tonali PA, Ricci E, Mirabella M. Broccolini A, et al. Among authors: mirabella m. J Neurochem. 2006 Feb;96(3):777-89. doi: 10.1111/j.1471-4159.2005.03584.x. Epub 2006 Jan 9. J Neurochem. 2006. PMID: 16405511 Free article.

5

NCAM is hyposialylated in hereditary inclusion body myopathy due to GNE mutations.

Ricci E, Broccolini A, Gidaro T, Morosetti R, Gliubizzi C, Frusciante R, Di Lella GM, Tonali PA, Mirabella M. Ricci E, et al. Among authors: mirabella m. Neurology. 2006 Mar 14;66(5):755-8. doi: 10.1212/01.wnl.0000200956.76449.3f. Neurology. 2006. PMID: 16534119

6

Hyposialylation of neprilysin possibly affects its expression and enzymatic activity in hereditary inclusion-body myopathy muscle.

Broccolini A, Gidaro T, De Cristofaro R, Morosetti R, Gliubizzi C, Ricci E, Tonali PA, Mirabella M. Broccolini A, et al. Among authors: mirabella m. J Neurochem. 2008 May;105(3):971-81. doi: 10.1111/j.1471-4159.2007.05208.x. Epub 2007 Dec 24. J Neurochem. 2008. PMID: 18182043 Free article.

7

Increased aging in primary muscle cultures of sporadic inclusion-body myositis.

Morosetti R, Broccolini A, Sancricca C, Gliubizzi C, Gidaro T, Tonali PA, Ricci E, Mirabella M. Morosetti R, et al. Among authors: mirabella m. Neurobiol Aging. 2010 Jul;31(7):1205-14. doi: 10.1016/j.neurobiolaging.2008.08.011. Epub 2008 Sep 26. Neurobiol Aging. 2010. PMID: 18823681

8

Hereditary inclusion-body myopathy: clues on pathogenesis and possible therapy.

Broccolini A, Gidaro T, Morosetti R, Mirabella M. Broccolini A, et al. Among authors: mirabella m. Muscle Nerve. 2009 Sep;40(3):340-9. doi: 10.1002/mus.21385. Muscle Nerve. 2009. PMID: 19618441 Review.

9

Analysis of NCAM helps identify unusual phenotypes of hereditary inclusion-body myopathy.

Broccolini A, Gidaro T, Tasca G, Morosetti R, Rodolico C, Ricci E, Mirabella M. Broccolini A, et al. Among authors: mirabella m. Neurology. 2010 Jul 20;75(3):265-72. doi: 10.1212/WNL.0b013e3181e8e8f1. Neurology. 2010. PMID: 20644153

10

Mesoangioblasts from facioscapulohumeral muscular dystrophy display in vivo a variable myogenic ability predictable by their in vitro behavior.

Morosetti R, Gidaro T, Broccolini A, Gliubizzi C, Sancricca C, Tonali PA, Ricci E, Mirabella M. Morosetti R, et al. Among authors: mirabella m. Cell Transplant. 2011;20(8):1299-313. doi: 10.3727/096368910X546571. Epub 2010 Dec 22. Cell Transplant. 2011. PMID: 21176400 Free article.

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