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Insights into the pathogenicity of rare missense GCK variants from the identification and functional characterization of compound heterozygous and double mutations inherited in cis.
Diabetes Care. 2012 Jul;35(7):1482-4. doi: 10.2337/dc11-2420. Epub 2012 May 18.
Diabetes Care. 2012.
PMID: 22611063
Free PMC article.
Use of HbA1c in the identification of patients with hyperglycaemia caused by a glucokinase mutation: observational case control studies.
Steele AM, Wensley KJ, Ellard S, Murphy R, Shepherd M, Colclough K, Hattersley AT, Shields BM.
Steele AM, et al. Among authors: wensley kj.
PLoS One. 2013 Jun 14;8(6):e65326. doi: 10.1371/journal.pone.0065326. Print 2013.
PLoS One. 2013.
PMID: 23799006
Free PMC article.
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The previously reported T342P GCK missense variant is not a pathogenic mutation causing MODY.
Steele AM, Tribble ND, Caswell R, Wensley KJ, Hattersley AT, Gloyn AL, Ellard S.
Steele AM, et al. Among authors: wensley kj.
Diabetologia. 2011 Aug;54(8):2202-5. doi: 10.1007/s00125-011-2194-5. Epub 2011 May 21.
Diabetologia. 2011.
PMID: 21604084
Free article.
No abstract available.
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Prevalence of vascular complications among patients with glucokinase mutations and prolonged, mild hyperglycemia.
Steele AM, Shields BM, Wensley KJ, Colclough K, Ellard S, Hattersley AT.
Steele AM, et al. Among authors: wensley kj.
JAMA. 2014 Jan 15;311(3):279-86. doi: 10.1001/jama.2013.283980.
JAMA. 2014.
PMID: 24430320
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Markers of β-Cell Failure Predict Poor Glycemic Response to GLP-1 Receptor Agonist Therapy in Type 2 Diabetes.
Jones AG, McDonald TJ, Shields BM, Hill AV, Hyde CJ, Knight BA, Hattersley AT; PRIBA Study Group.
Jones AG, et al.
Diabetes Care. 2016 Feb;39(2):250-7. doi: 10.2337/dc15-0258. Epub 2015 Aug 4.
Diabetes Care. 2016.
PMID: 26242184
Free PMC article.
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