Ellenrieder V - Search Results

1

GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression.

Baumgart S, Chen NM, Zhang JS, Billadeau DD, Gaisina IN, Kozikowski AP, Singh SK, Fink D, Ströbel P, Klindt C, Zhang L, Bamlet WR, Koenig A, Hessmann E, Gress TM, Ellenrieder V, Neesse A. Baumgart S, et al. Among authors: ellenrieder v. Mol Cancer Ther. 2016 Mar;15(3):491-502. doi: 10.1158/1535-7163.MCT-15-0309. Epub 2016 Jan 28. Mol Cancer Ther. 2016. PMID: 26823495 Free PMC article.

2

Role of extracellular matrix in pancreatic diseases.

Gress TM, Menke A, Bachem M, Müller-Pillasch F, Ellenrieder V, Weidenbach H, Wagner M, Adler G. Gress TM, et al. Among authors: ellenrieder v. Digestion. 1998 Nov-Dec;59(6):625-37. doi: 10.1159/000007567. Digestion. 1998. PMID: 9813385 Free article. Review. No abstract available.

3

Expression and differential regulation of connective tissue growth factor in pancreatic cancer cells.

Wenger C, Ellenrieder V, Alber B, Lacher U, Menke A, Hameister H, Wilda M, Iwamura T, Beger HG, Adler G, Gress TM. Wenger C, et al. Among authors: ellenrieder v. Oncogene. 1999 Jan 28;18(4):1073-80. doi: 10.1038/sj.onc.1202395. Oncogene. 1999. PMID: 10023684

4

Invasion and metastasis in pancreatic cancer.

Ellenrieder V, Adler G, Gress TM. Ellenrieder V, et al. Ann Oncol. 1999;10 Suppl 4:46-50. Ann Oncol. 1999. PMID: 10436784 Review.

5

Use of representational difference analysis to study the effect of TGFB on the expression profile of a pancreatic cancer cell line.

Geng MM, Ellenrieder V, Wallrapp C, Müller-Pillasch F, Sommer G, Adler G, Gress TM. Geng MM, et al. Among authors: ellenrieder v. Genes Chromosomes Cancer. 1999 Sep;26(1):70-9. doi: 10.1002/(sici)1098-2264(199909)26:1<70::aid-gcc10>3.0.co;2-a. Genes Chromosomes Cancer. 1999. PMID: 10441008

6

Role of MT-MMPs and MMP-2 in pancreatic cancer progression.

Ellenrieder V, Alber B, Lacher U, Hendler SF, Menke A, Boeck W, Wagner M, Wilda M, Friess H, Büchler M, Adler G, Gress TM. Ellenrieder V, et al. Int J Cancer. 2000 Jan 1;85(1):14-20. doi: 10.1002/(sici)1097-0215(20000101)85:1<14::aid-ijc3>3.0.co;2-o. Int J Cancer. 2000. PMID: 10585576 Free article.

7

Transforming growth factor beta1 treatment leads to an epithelial-mesenchymal transdifferentiation of pancreatic cancer cells requiring extracellular signal-regulated kinase 2 activation.

Ellenrieder V, Hendler SF, Boeck W, Seufferlein T, Menke A, Ruhland C, Adler G, Gress TM. Ellenrieder V, et al. Cancer Res. 2001 May 15;61(10):4222-8. Cancer Res. 2001. PMID: 11358848

8

TGF-beta-induced invasiveness of pancreatic cancer cells is mediated by matrix metalloproteinase-2 and the urokinase plasminogen activator system.

Ellenrieder V, Hendler SF, Ruhland C, Boeck W, Adler G, Gress TM. Ellenrieder V, et al. Int J Cancer. 2001 Jul 15;93(2):204-11. doi: 10.1002/ijc.1330. Int J Cancer. 2001. PMID: 11410867 Free article.

9

A conserved alpha-helical motif mediates the interaction of Sp1-like transcriptional repressors with the corepressor mSin3A.

Zhang JS, Moncrieffe MC, Kaczynski J, Ellenrieder V, Prendergast FG, Urrutia R. Zhang JS, et al. Among authors: ellenrieder v. Mol Cell Biol. 2001 Aug;21(15):5041-9. doi: 10.1128/MCB.21.15.5041-5049.2001. Mol Cell Biol. 2001. PMID: 11438660 Free PMC article.

10

The Sp1-like protein BTEB3 inhibits transcription via the basic transcription element box by interacting with mSin3A and HDAC-1 co-repressors and competing with Sp1.

Kaczynski J, Zhang JS, Ellenrieder V, Conley A, Duenes T, Kester H, van Der Burg B, Urrutia R. Kaczynski J, et al. Among authors: ellenrieder v. J Biol Chem. 2001 Sep 28;276(39):36749-56. doi: 10.1074/jbc.M105831200. Epub 2001 Jul 27. J Biol Chem. 2001. PMID: 11477107 Free article.

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