Mair N - Search Results

1

Changes in Ionic Conductance Signature of Nociceptive Neurons Underlying Fabry Disease Phenotype.

Namer B, Ørstavik K, Schmidt R, Mair N, Kleggetveit IP, Zeidler M, Martha T, Jorum E, Schmelz M, Kalpachidou T, Kress M, Langeslag M. Namer B, et al. Among authors: mair n. Front Neurol. 2017 Jul 14;8:335. doi: 10.3389/fneur.2017.00335. eCollection 2017. Front Neurol. 2017. PMID: 28769867 Free PMC article.

2

Endogenous tumor necrosis factor alpha (TNFalpha) requires TNF receptor type 2 to generate heat hyperalgesia in a mouse cancer model.

Constantin CE, Mair N, Sailer CA, Andratsch M, Xu ZZ, Blumer MJ, Scherbakov N, Davis JB, Bluethmann H, Ji RR, Kress M. Constantin CE, et al. Among authors: mair n. J Neurosci. 2008 May 7;28(19):5072-81. doi: 10.1523/JNEUROSCI.4476-07.2008. J Neurosci. 2008. PMID: 18463260 Free PMC article.

3

A key role for gp130 expressed on peripheral sensory nerves in pathological pain.

Andratsch M, Mair N, Constantin CE, Scherbakov N, Benetti C, Quarta S, Vogl C, Sailer CA, Uceyler N, Brockhaus J, Martini R, Sommer C, Zeilhofer HU, Müller W, Kuner R, Davis JB, Rose-John S, Kress M. Andratsch M, et al. Among authors: mair n. J Neurosci. 2009 Oct 28;29(43):13473-83. doi: 10.1523/JNEUROSCI.1822-09.2009. J Neurosci. 2009. PMID: 19864560 Free PMC article.

4

Genetic evidence for involvement of neuronally expressed S1P₁ receptor in nociceptor sensitization and inflammatory pain.

Mair N, Benetti C, Andratsch M, Leitner MG, Constantin CE, Camprubí-Robles M, Quarta S, Biasio W, Kuner R, Gibbins IL, Kress M, Haberberger RV. Mair N, et al. PLoS One. 2011 Feb 17;6(2):e17268. doi: 10.1371/journal.pone.0017268. PLoS One. 2011. PMID: 21359147 Free PMC article.

5

Oncostatin M induces heat hypersensitivity by gp130-dependent sensitization of TRPV1 in sensory neurons.

Langeslag M, Constantin CE, Andratsch M, Quarta S, Mair N, Kress M. Langeslag M, et al. Among authors: mair n. Mol Pain. 2011 Dec 23;7:102. doi: 10.1186/1744-8069-7-102. Mol Pain. 2011. PMID: 22196363 Free PMC article.

6

Sphingosine-1-phosphate-induced nociceptor excitation and ongoing pain behavior in mice and humans is largely mediated by S1P3 receptor.

Camprubí-Robles M, Mair N, Andratsch M, Benetti C, Beroukas D, Rukwied R, Langeslag M, Proia RL, Schmelz M, Ferrer Montiel AV, Haberberger RV, Kress M. Camprubí-Robles M, et al. Among authors: mair n. J Neurosci. 2013 Feb 6;33(6):2582-92. doi: 10.1523/JNEUROSCI.4479-12.2013. J Neurosci. 2013. PMID: 23392686 Free PMC article. Clinical Trial.

7

Hypoxia-inducible factor 1 regulates heat and cold pain sensitivity and persistence.

Kanngiesser M, Mair N, Lim HY, Zschiebsch K, Blees J, Häussler A, Brüne B, Ferreiròs N, Kress M, Tegeder I. Kanngiesser M, et al. Among authors: mair n. Antioxid Redox Signal. 2014 Jun 1;20(16):2555-71. doi: 10.1089/ars.2013.5494. Epub 2014 Jan 30. Antioxid Redox Signal. 2014. PMID: 24144405

8

Sphingosine 1-phosphate to p38 signaling via S1P1 receptor and Gαi/o evokes augmentation of capsaicin-induced ionic currents in mouse sensory neurons.

Langeslag M, Quarta S, Leitner MG, Kress M, Mair N. Langeslag M, et al. Among authors: mair n. Mol Pain. 2014 Nov 28;10:74. doi: 10.1186/1744-8069-10-74. Mol Pain. 2014. PMID: 25431213 Free PMC article.

9

Identification of Chloride Channels CLCN3 and CLCN5 Mediating the Excitatory Cl^- Currents Activated by Sphingosine-1-Phosphate in Sensory Neurons.

Qi Y, Mair N, Kummer KK, Leitner MG, Camprubí-Robles M, Langeslag M, Kress M. Qi Y, et al. Among authors: mair n. Front Mol Neurosci. 2018 Feb 9;11:33. doi: 10.3389/fnmol.2018.00033. eCollection 2018. Front Mol Neurosci. 2018. PMID: 29479306 Free PMC article.

10

Impaired mechanical, heat, and cold nociception in a murine model of genetic TACE/ADAM17 knockdown.

Quarta S, Mitrić M, Kalpachidou T, Mair N, Schiefermeier-Mach N, Andratsch M, Qi Y, Langeslag M, Malsch P, Rose-John S, Kress M. Quarta S, et al. Among authors: mair n. FASEB J. 2019 Mar;33(3):4418-4431. doi: 10.1096/fj.201801901R. Epub 2018 Dec 26. FASEB J. 2019. PMID: 30586315 Free PMC article.

Our data suggest that ADAM17 contributes to the processing of painful stimuli, with a complex mode of action orchestrating the function of neurons along the pain pathway.-Quarta, S., Mitric, M., Kalpachidou, T., Mair, N., Schiefermeier-Mach, N., Andratsch, M. …

Our data suggest that ADAM17 contributes to the processing of painful stimuli, with a complex mode of action orchestrating the function of n …

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