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Functional non-synonymous variants of ABCG2 and gout risk.
Stiburkova B, Pavelcova K, Zavada J, Petru L, Simek P, Cepek P, Pavlikova M, Matsuo H, Merriman TR, Pavelka K. Stiburkova B, et al. Among authors: matsuo h. Rheumatology (Oxford). 2017 Nov 1;56(11):1982-1992. doi: 10.1093/rheumatology/kex295. Rheumatology (Oxford). 2017. PMID: 28968913
Common defects of ABCG2, a high-capacity urate exporter, cause gout: a function-based genetic analysis in a Japanese population.
Matsuo H, Takada T, Ichida K, Nakamura T, Nakayama A, Ikebuchi Y, Ito K, Kusanagi Y, Chiba T, Tadokoro S, Takada Y, Oikawa Y, Inoue H, Suzuki K, Okada R, Nishiyama J, Domoto H, Watanabe S, Fujita M, Morimoto Y, Naito M, Nishio K, Hishida A, Wakai K, Asai Y, Niwa K, Kamakura K, Nonoyama S, Sakurai Y, Hosoya T, Kanai Y, Suzuki H, Hamajima N, Shinomiya N. Matsuo H, et al. Sci Transl Med. 2009 Nov 4;1(5):5ra11. doi: 10.1126/scitranslmed.3000237. Sci Transl Med. 2009. PMID: 20368174
Identification of ABCG2 dysfunction as a major factor contributing to gout.
Matsuo H, Takada T, Ichida K, Nakamura T, Nakayama A, Takada Y, Okada C, Sakurai Y, Hosoya T, Kanai Y, Suzuki H, Shinomiya N. Matsuo H, et al. Nucleosides Nucleotides Nucleic Acids. 2011 Dec;30(12):1098-104. doi: 10.1080/15257770.2011.627902. Nucleosides Nucleotides Nucleic Acids. 2011. PMID: 22132963
ABCG2/BCRP dysfunction as a major cause of gout.
Matsuo H, Takada T, Ichida K, Nakamura T, Nakayama A, Suzuki H, Hosoya T, Shinomiya N. Matsuo H, et al. Nucleosides Nucleotides Nucleic Acids. 2011 Dec;30(12):1117-28. doi: 10.1080/15257770.2011.633954. Nucleosides Nucleotides Nucleic Acids. 2011. PMID: 22132966
Common dysfunctional variants in ABCG2 are a major cause of early-onset gout.
Matsuo H, Ichida K, Takada T, Nakayama A, Nakashima H, Nakamura T, Kawamura Y, Takada Y, Yamamoto K, Inoue H, Oikawa Y, Naito M, Hishida A, Wakai K, Okada C, Shimizu S, Sakiyama M, Chiba T, Ogata H, Niwa K, Hosoyamada M, Mori A, Hamajima N, Suzuki H, Kanai Y, Sakurai Y, Hosoya T, Shimizu T, Shinomiya N. Matsuo H, et al. Sci Rep. 2013;3:2014. doi: 10.1038/srep02014. Sci Rep. 2013. PMID: 23774753 Free PMC article.
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overload.
Matsuo H, Nakayama A, Sakiyama M, Chiba T, Shimizu S, Kawamura Y, Nakashima H, Nakamura T, Takada Y, Oikawa Y, Takada T, Nakaoka H, Abe J, Inoue H, Wakai K, Kawai S, Guang Y, Nakagawa H, Ito T, Niwa K, Yamamoto K, Sakurai Y, Suzuki H, Hosoya T, Ichida K, Shimizu T, Shinomiya N. Matsuo H, et al. Sci Rep. 2014 Jan 20;4:3755. doi: 10.1038/srep03755. Sci Rep. 2014. PMID: 24441388 Free PMC article.
Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factors.
Nakayama A, Matsuo H, Nakaoka H, Nakamura T, Nakashima H, Takada Y, Oikawa Y, Takada T, Sakiyama M, Shimizu S, Kawamura Y, Chiba T, Abe J, Wakai K, Kawai S, Okada R, Tamura T, Shichijo Y, Akashi A, Suzuki H, Hosoya T, Sakurai Y, Ichida K, Shinomiya N. Nakayama A, et al. Among authors: matsuo h. Sci Rep. 2014 Jun 9;4:5227. doi: 10.1038/srep05227. Sci Rep. 2014. PMID: 24909660 Free PMC article.
ABCG2 dysfunction increases the risk of renal overload hyperuricemia.
Matsuo H, Takada T, Nakayama A, Shimizu T, Sakiyama M, Shimizu S, Chiba T, Nakashima H, Nakamura T, Takada Y, Sakurai Y, Hosoya T, Shinomiya N, Ichida K. Matsuo H, et al. Nucleosides Nucleotides Nucleic Acids. 2014;33(4-6):266-74. doi: 10.1080/15257770.2013.866679. Nucleosides Nucleotides Nucleic Acids. 2014. PMID: 24940678
2,264 results