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TLR9 stimulation of B-cells induces transcription of p53 and prevents spontaneous and irradiation-induced cell death independent of DNA damage responses. Implications for Common variable immunodeficiency.
PLoS One. 2017 Oct 3;12(10):e0185708. doi: 10.1371/journal.pone.0185708. eCollection 2017.
PLoS One. 2017.
PMID: 28973009
Free PMC article.
Retinoic acid improves defective TLR9/RP105-induced immune responses in common variable immunodeficiency-derived B cells.
Indrevær RL, Holm KL, Aukrust P, Osnes LT, Naderi EH, Fevang B, Blomhoff HK.
Indrevær RL, et al.
J Immunol. 2013 Oct 1;191(7):3624-33. doi: 10.4049/jimmunol.1300213. Epub 2013 Sep 4.
J Immunol. 2013.
PMID: 24006462
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IRF4 Is a Critical Gene in Retinoic Acid-Mediated Plasma Cell Formation and Is Deregulated in Common Variable Immunodeficiency-Derived B Cells.
Indrevær RL, Moskaug JØ, Paur I, Bøhn SK, Jørgensen SF, Blomhoff R, Aukrust P, Fevang B, Blomhoff HK.
Indrevær RL, et al.
J Immunol. 2015 Sep 15;195(6):2601-11. doi: 10.4049/jimmunol.1500250. Epub 2015 Aug 14.
J Immunol. 2015.
PMID: 26276871
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Myeloid cell leukaemia 1 has a vital role in retinoic acid-mediated protection of Toll-like receptor 9-stimulated B cells from spontaneous and DNA damage-induced apoptosis.
Holm KL, Indrevaer RL, Myklebust JH, Kolstad A, Moskaug JØ, Naderi EH, Blomhoff HK.
Holm KL, et al. Among authors: indrevaer rl.
Immunology. 2016 Sep;149(1):62-73. doi: 10.1111/imm.12629. Epub 2016 Jul 25.
Immunology. 2016.
PMID: 27278254
Free PMC article.
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TLR9-signaling is required for turning retinoic acid into a potent stimulator of RP105 (CD180)-mediated proliferation and IgG synthesis in human memory B cells.
Eriksen AB, Indrevær RL, Holm KL, Landskron J, Blomhoff HK.
Eriksen AB, et al. Among authors: indrevaer rl.
Cell Immunol. 2012 Sep;279(1):87-95. doi: 10.1016/j.cellimm.2012.09.003. Epub 2012 Sep 19.
Cell Immunol. 2012.
PMID: 23103284
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