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Adamts10 inactivation in mice leads to persistence of ocular microfibrils subsequent to reduced fibrillin-2 cleavage.
Matrix Biol. 2019 Apr;77:117-128. doi: 10.1016/j.matbio.2018.09.004. Epub 2018 Sep 7.
Matrix Biol. 2019.
PMID: 30201140
Free PMC article.
ADAMTS10 protein interacts with fibrillin-1 and promotes its deposition in extracellular matrix of cultured fibroblasts.
Kutz WE, Wang LW, Bader HL, Majors AK, Iwata K, Traboulsi EI, Sakai LY, Keene DR, Apte SS.
Kutz WE, et al.
J Biol Chem. 2011 May 13;286(19):17156-67. doi: 10.1074/jbc.M111.231571. Epub 2011 Mar 14.
J Biol Chem. 2011.
PMID: 21402694
Free PMC article.
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Functional analysis of an ADAMTS10 signal peptide mutation in Weill-Marchesani syndrome demonstrates a long-range effect on secretion of the full-length enzyme.
Kutz WE, Wang LW, Dagoneau N, Odrcic KJ, Cormier-Daire V, Traboulsi EI, Apte SS.
Kutz WE, et al.
Hum Mutat. 2008 Dec;29(12):1425-34. doi: 10.1002/humu.20797.
Hum Mutat. 2008.
PMID: 18567016
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WISP3, the gene responsible for the human skeletal disease progressive pseudorheumatoid dysplasia, is not essential for skeletal function in mice.
Kutz WE, Gong Y, Warman ML.
Kutz WE, et al.
Mol Cell Biol. 2005 Jan;25(1):414-21. doi: 10.1128/MCB.25.1.414-421.2005.
Mol Cell Biol. 2005.
PMID: 15601861
Free PMC article.
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Normal growth and development in mice over-expressing the CCN family member WISP3.
Nakamura Y, Cui Y, Fernando C, Kutz WE, Warman ML.
Nakamura Y, et al. Among authors: kutz we.
J Cell Commun Signal. 2009 Jun;3(2):105-13. doi: 10.1007/s12079-009-0040-z. Epub 2009 Apr 29.
J Cell Commun Signal. 2009.
PMID: 19401829
Free PMC article.
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