Rotter V - Search Results

1

Corrigendum to: The role of p53 in base excision repair following genotoxic stress.

Zurer I, Hofseth LJ, Cohen Y, Xu-Welliver M, Hussain SP, Harris CC, Rotter V. Zurer I, et al. Among authors: rotter v. Carcinogenesis. 2021 Jun 21;42(6):903. doi: 10.1093/carcin/bgab028. Carcinogenesis. 2021. PMID: 33991083 Free PMC article. No abstract available.

2

The role of p53 in base excision repair following genotoxic stress.

Zurer I, Hofseth LJ, Cohen Y, Xu-Welliver M, Hussain SP, Harris CC, Rotter V. Zurer I, et al. Among authors: rotter v. Carcinogenesis. 2004 Jan;25(1):11-9. doi: 10.1093/carcin/bgg186. Epub 2003 Oct 10. Carcinogenesis. 2004. PMID: 14555612

3

The onset of p53-dependent DNA repair or apoptosis is determined by the level of accumulated damaged DNA.

Offer H, Erez N, Zurer I, Tang X, Milyavsky M, Goldfinger N, Rotter V. Offer H, et al. Among authors: rotter v. Carcinogenesis. 2002 Jun;23(6):1025-32. doi: 10.1093/carcin/23.6.1025. Carcinogenesis. 2002. PMID: 12082025

4

Nitric oxide-induced cellular stress and p53 activation in chronic inflammation.

Hofseth LJ, Saito S, Hussain SP, Espey MG, Miranda KM, Araki Y, Jhappan C, Higashimoto Y, He P, Linke SP, Quezado MM, Zurer I, Rotter V, Wink DA, Appella E, Harris CC. Hofseth LJ, et al. Among authors: rotter v. Proc Natl Acad Sci U S A. 2003 Jan 7;100(1):143-8. doi: 10.1073/pnas.0237083100. Epub 2002 Dec 23. Proc Natl Acad Sci U S A. 2003. PMID: 12518062 Free PMC article.

5

The adaptive imbalance in base excision-repair enzymes generates microsatellite instability in chronic inflammation.

Hofseth LJ, Khan MA, Ambrose M, Nikolayeva O, Xu-Welliver M, Kartalou M, Hussain SP, Roth RB, Zhou X, Mechanic LE, Zurer I, Rotter V, Samson LD, Harris CC. Hofseth LJ, et al. Among authors: rotter v. J Clin Invest. 2003 Dec;112(12):1887-94. doi: 10.1172/JCI19757. J Clin Invest. 2003. PMID: 14679184 Free PMC article.

6

p53 modulates base excision repair activity in a cell cycle-specific manner after genotoxic stress.

Offer H, Zurer I, Banfalvi G, Reha'k M, Falcovitz A, Milyavsky M, Goldfinger N, Rotter V. Offer H, et al. Among authors: rotter v. Cancer Res. 2001 Jan 1;61(1):88-96. Cancer Res. 2001. PMID: 11196204

7

Transcriptional programs following genetic alterations in p53, INK4A, and H-Ras genes along defined stages of malignant transformation.

Milyavsky M, Tabach Y, Shats I, Erez N, Cohen Y, Tang X, Kalis M, Kogan I, Buganim Y, Goldfinger N, Ginsberg D, Harris CC, Domany E, Rotter V. Milyavsky M, et al. Among authors: rotter v. Cancer Res. 2005 Jun 1;65(11):4530-43. doi: 10.1158/0008-5472.CAN-04-3880. Cancer Res. 2005. PMID: 15930270

8

COOH-terminal domain of p53 modulates p53-mediated transcriptional transactivation, cell growth, and apoptosis.

Zhou X, Wang XW, Xu L, Hagiwara K, Nagashima M, Wolkowicz R, Zurer I, Rotter V, Harris CC. Zhou X, et al. Among authors: rotter v. Cancer Res. 1999 Feb 15;59(4):843-8. Cancer Res. 1999. PMID: 10029073

9

Structural and functional involvement of p53 in BER in vitro and in vivo.

Offer H, Milyavsky M, Erez N, Matas D, Zurer I, Harris CC, Rotter V. Offer H, et al. Among authors: rotter v. Oncogene. 2001 Feb 1;20(5):581-9. doi: 10.1038/sj.onc.1204120. Oncogene. 2001. PMID: 11313990

10

Transactivation of the EGR1 gene contributes to mutant p53 gain of function.

Weisz L, Zalcenstein A, Stambolsky P, Cohen Y, Goldfinger N, Oren M, Rotter V. Weisz L, et al. Among authors: rotter v. Cancer Res. 2004 Nov 15;64(22):8318-27. doi: 10.1158/0008-5472.CAN-04-1145. Cancer Res. 2004. PMID: 15548700 Retracted.

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