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Linking ATP and allosteric sites to achieve superadditive binding with bivalent EGFR kinase inhibitors.
Commun Chem. 2024 Feb 20;7(1):38. doi: 10.1038/s42004-024-01108-3.
Commun Chem. 2024.
PMID: 38378740
Free PMC article.
Linking ATP and allosteric sites to achieve superadditive binding with bivalent EGFR kinase inhibitors.
Heppner D, Wittlinger F, Ogboo B, Shevchenko E, Damghani T, Pham C, Schaeffner I, Oligny B, Chitnis S, Beyett T, Rasch A, Buckley B, Urul D, Shaurova T, May E, Schaefer E, Eck M, Hershberger P, Poso A, Laufer S.
Heppner D, et al.
Res Sq [Preprint]. 2023 Sep 13:rs.3.rs-3286949. doi: 10.21203/rs.3.rs-3286949/v1.
Res Sq. 2023.
PMID: 37790373
Free PMC article.
Updated.
Preprint.
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Inhibition of a lower potency target drives the anticancer activity of a clinical p38 inhibitor.
Bhattacharjee D, Bakar J, Chitnis SP, Sausville EL, Ashtekar KD, Mendelson BE, Long K, Smith JC, Heppner DE, Sheltzer JM.
Bhattacharjee D, et al. Among authors: chitnis sp.
Cell Chem Biol. 2023 Oct 19;30(10):1211-1222.e5. doi: 10.1016/j.chembiol.2023.09.013. Epub 2023 Oct 11.
Cell Chem Biol. 2023.
PMID: 37827156
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