Shurtleff SA - Search Results

1

TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis.

Shurtleff SA, Buijs A, Behm FG, Rubnitz JE, Raimondi SC, Hancock ML, Chan GC, Pui CH, Grosveld G, Downing JR. Shurtleff SA, et al. Leukemia. 1995 Dec;9(12):1985-9. Leukemia. 1995. PMID: 8609706

2

A complex variant t(8;21) involving chromosome 3 in a child with acute myeloblastic leukemia with eosinophilia (AML M4Eo).

Mathew S, Shurtleff S, Ribeiro RC, Behm FG, Raimondi SC. Mathew S, et al. Leuk Lymphoma. 2003 Jan;44(1):183-7. doi: 10.1080/1042819031000057289. Leuk Lymphoma. 2003. PMID: 12691161 Review.

3

Reassessment of the prognostic significance of hypodiploidy in pediatric patients with acute lymphoblastic leukemia.

Raimondi SC, Zhou Y, Mathew S, Shurtleff SA, Sandlund JT, Rivera GK, Behm FG, Pui CH. Raimondi SC, et al. Among authors: shurtleff sa. Cancer. 2003 Dec 15;98(12):2715-22. doi: 10.1002/cncr.11841. Cancer. 2003. PMID: 14669294 Free article.

4

Near-triploidy and near-tetraploidy in childhood acute lymphoblastic leukemia: association with B-lineage blast cells carrying the ETV6-RUNX1 fusion, T-lineage immunophenotype, and favorable outcome.

Raimondi SC, Zhou Y, Shurtleff SA, Rubnitz JE, Pui CH, Behm FG. Raimondi SC, et al. Among authors: shurtleff sa. Cancer Genet Cytogenet. 2006 Aug;169(1):50-7. doi: 10.1016/j.cancergencyto.2006.04.006. Cancer Genet Cytogenet. 2006. PMID: 16875937

5

ETV6-RUNX1-positive childhood acute lymphoblastic leukemia: improved outcome with contemporary therapy.

Bhojwani D, Pei D, Sandlund JT, Jeha S, Ribeiro RC, Rubnitz JE, Raimondi SC, Shurtleff S, Onciu M, Cheng C, Coustan-Smith E, Bowman WP, Howard SC, Metzger ML, Inaba H, Leung W, Evans WE, Campana D, Relling MV, Pui CH. Bhojwani D, et al. Leukemia. 2012 Feb;26(2):265-70. doi: 10.1038/leu.2011.227. Epub 2011 Aug 26. Leukemia. 2012. PMID: 21869842 Free PMC article.

6

The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription.

Hiebert SW, Sun W, Davis JN, Golub T, Shurtleff S, Buijs A, Downing JR, Grosveld G, Roussell MF, Gilliland DG, Lenny N, Meyers S. Hiebert SW, et al. Mol Cell Biol. 1996 Apr;16(4):1349-55. doi: 10.1128/MCB.16.4.1349. Mol Cell Biol. 1996. PMID: 8657108 Free PMC article.

7

MN1 overexpression is an important step in the development of inv(16) AML.

Carella C, Bonten J, Sirma S, Kranenburg TA, Terranova S, Klein-Geltink R, Shurtleff S, Downing JR, Zwarthoff EC, Liu PP, Grosveld GC. Carella C, et al. Leukemia. 2007 Aug;21(8):1679-90. doi: 10.1038/sj.leu.2404778. Epub 2007 May 24. Leukemia. 2007. PMID: 17525718

8

Frequent deletion of p16INK4a/MTS1 and p15INK4b/MTS2 in pediatric acute lymphoblastic leukemia.

Okuda T, Shurtleff SA, Valentine MB, Raimondi SC, Head DR, Behm F, Curcio-Brint AM, Liu Q, Pui CH, Sherr CJ, et al. Okuda T, et al. Among authors: shurtleff sa. Blood. 1995 May 1;85(9):2321-30. Blood. 1995. PMID: 7727766 Free article.

9

Heterogeneity in CBF beta/MYH11 fusion messages encoded by the inv(16)(p13q22) and the t(16;16)(p13;q22) in acute myelogenous leukemia.

Shurtleff SA, Meyers S, Hiebert SW, Raimondi SC, Head DR, Willman CL, Wolman S, Slovak ML, Carroll AJ, Behm F, et al. Shurtleff SA, et al. Blood. 1995 Jun 15;85(12):3695-703. Blood. 1995. PMID: 7780153 Free article.

10

11q23/MLL rearrangement confers a poor prognosis in infants with acute lymphoblastic leukemia.

Pui CH, Behm FG, Downing JR, Hancock ML, Shurtleff SA, Ribeiro RC, Head DR, Mahmoud HH, Sandlund JT, Furman WL, et al. Pui CH, et al. Among authors: shurtleff sa. J Clin Oncol. 1994 May;12(5):909-15. doi: 10.1200/JCO.1994.12.5.909. J Clin Oncol. 1994. PMID: 8164041

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