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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
---|---|
2008 | 1 |
2009 | 2 |
2013 | 2 |
2018 | 1 |
2024 | 0 |
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6 results
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Page 1
Inactivating hepatic follistatin alleviates hyperglycemia.
Nat Med. 2018 Jul;24(7):1058-1069. doi: 10.1038/s41591-018-0048-0. Epub 2018 Jun 4.
Nat Med. 2018.
PMID: 29867232
Free PMC article.
Foxo1 integrates insulin signaling with mitochondrial function in the liver.
Cheng Z, Guo S, Copps K, Dong X, Kollipara R, Rodgers JT, Depinho RA, Puigserver P, White MF.
Cheng Z, et al.
Nat Med. 2009 Nov;15(11):1307-11. doi: 10.1038/nm.2049. Epub 2009 Oct 18.
Nat Med. 2009.
PMID: 19838201
Free PMC article.
Item in Clipboard
Genetic inactivation of pyruvate dehydrogenase kinases improves hepatic insulin resistance induced diabetes.
Tao R, Xiong X, Harris RA, White MF, Dong XC.
Tao R, et al.
PLoS One. 2013 Aug 5;8(8):e71997. doi: 10.1371/journal.pone.0071997. Print 2013.
PLoS One. 2013.
PMID: 23940800
Free PMC article.
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Deletion of hepatic FoxO1/3/4 genes in mice significantly impacts on glucose metabolism through downregulation of gluconeogenesis and upregulation of glycolysis.
Xiong X, Tao R, DePinho RA, Dong XC.
Xiong X, et al.
PLoS One. 2013 Aug 28;8(8):e74340. doi: 10.1371/journal.pone.0074340. eCollection 2013.
PLoS One. 2013.
PMID: 24015318
Free PMC article.
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Inactivation of hepatic Foxo1 by insulin signaling is required for adaptive nutrient homeostasis and endocrine growth regulation.
Dong XC, Copps KD, Guo S, Li Y, Kollipara R, DePinho RA, White MF.
Dong XC, et al.
Cell Metab. 2008 Jul;8(1):65-76. doi: 10.1016/j.cmet.2008.06.006.
Cell Metab. 2008.
PMID: 18590693
Free PMC article.
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The Irs1 branch of the insulin signaling cascade plays a dominant role in hepatic nutrient homeostasis.
Guo S, Copps KD, Dong X, Park S, Cheng Z, Pocai A, Rossetti L, Sajan M, Farese RV, White MF.
Guo S, et al.
Mol Cell Biol. 2009 Sep;29(18):5070-83. doi: 10.1128/MCB.00138-09. Epub 2009 Jul 13.
Mol Cell Biol. 2009.
PMID: 19596788
Free PMC article.
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