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Oral treatment with Cu(II)(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis.
Roberts BR, Lim NK, McAllum EJ, Donnelly PS, Hare DJ, Doble PA, Turner BJ, Price KA, Lim SC, Paterson BM, Hickey JL, Rhoads TW, Williams JR, Kanninen KM, Hung LW, Liddell JR, Grubman A, Monty JF, Llanos RM, Kramer DR, Mercer JF, Bush AI, Masters CL, Duce JA, Li QX, Beckman JS, Barnham KJ, White AR, Crouch PJ. Roberts BR, et al. Among authors: beckman js. J Neurosci. 2014 Jun 4;34(23):8021-31. doi: 10.1523/JNEUROSCI.4196-13.2014. J Neurosci. 2014. PMID: 24899723 Free PMC article.
Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SOD(G93A) mice co-expressing the Copper-Chaperone-for-SOD.
Williams JR, Trias E, Beilby PR, Lopez NI, Labut EM, Bradford CS, Roberts BR, McAllum EJ, Crouch PJ, Rhoads TW, Pereira C, Son M, Elliott JL, Franco MC, Estévez AG, Barbeito L, Beckman JS. Williams JR, et al. Among authors: beckman js. Neurobiol Dis. 2016 May;89:1-9. doi: 10.1016/j.nbd.2016.01.020. Epub 2016 Jan 27. Neurobiol Dis. 2016. PMID: 26826269 Free PMC article.
Superoxide dismutase and the death of motoneurons in ALS.
Beckman JS, Estévez AG, Crow JP, Barbeito L. Beckman JS, et al. Trends Neurosci. 2001 Nov;24(11 Suppl):S15-20. doi: 10.1016/s0166-2236(00)01981-0. Trends Neurosci. 2001. PMID: 11881740 Review.
201 results