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A candidate gene approach identifies an IL33 genetic variant as a novel genetic risk factor for GCA.
Márquez A, Solans R, Hernández-Rodríguez J, Cid MC, Castañeda S, Ramentol M, Rodriguez-Rodriguez L, Narváez J, Blanco R, Ortego-Centeno N; Spanish GCA Consortium; Palm O, Diamantopoulos AP, Braun N, Moosig F, Witte T, Beretta L, Lunardi C, Cimmino MA, Vaglio A, Salvarani C, González-Gay MA, Martín J. Márquez A, et al. Among authors: cid mc. PLoS One. 2014 Nov 19;9(11):e113476. doi: 10.1371/journal.pone.0113476. eCollection 2014. PLoS One. 2014. PMID: 25409453 Free PMC article.
Circulating soluble adhesion molecules in patients with giant cell arteritis. Correlation between soluble intercellular adhesion molecule-1 (sICAM-1) concentrations and disease activity.
Coll-Vinent B, Vilardell C, Font C, Oristrell J, Hernández-Rodríguez J, Yagüe J, Urbano-Márquez A, Grau JM, Cid MC. Coll-Vinent B, et al. Among authors: cid mc. Ann Rheum Dis. 1999 Mar;58(3):189-92. doi: 10.1136/ard.58.3.189. Ann Rheum Dis. 1999. PMID: 10364919 Free PMC article.
Cell adhesion molecules in the development of inflammatory infiltrates in giant cell arteritis: inflammation-induced angiogenesis as the preferential site of leukocyte-endothelial cell interactions.
Cid MC, Cebrián M, Font C, Coll-Vinent B, Hernández-Rodríguez J, Esparza J, Urbano-Márquez A, Grau JM. Cid MC, et al. Arthritis Rheum. 2000 Jan;43(1):184-94. doi: 10.1002/1529-0131(200001)43:1<184::AID-ANR23>3.0.CO;2-N. Arthritis Rheum. 2000. PMID: 10643715 Free article.
Small-vessel vasculitis surrounding a spared temporal artery: clinical and pathological findings in a series of twenty-eight patients.
Esteban MJ, Font C, Hernández-Rodríguez J, Valls-Solé J, Sanmartí R, Cardellach F, García-Martínez A, Campo E, Urbano-Márquez A, Grau JM, Cid MC. Esteban MJ, et al. Among authors: cid mc. Arthritis Rheum. 2001 Jun;44(6):1387-95. doi: 10.1002/1529-0131(200106)44:6<1387::AID-ART232>3.0.CO;2-B. Arthritis Rheum. 2001. PMID: 11407699 Free article.
A strong initial systemic inflammatory response is associated with higher corticosteroid requirements and longer duration of therapy in patients with giant-cell arteritis.
Hernández-Rodríguez J, García-Martínez A, Casademont J, Filella X, Esteban MJ, López-Soto A, Fernández-Solà J, Urbano-Márquez A, Grau JM, Cid MC. Hernández-Rodríguez J, et al. Among authors: cid mc. Arthritis Rheum. 2002 Feb;47(1):29-35. doi: 10.1002/art1.10161. Arthritis Rheum. 2002. PMID: 11932875 Free article.
Elevated production of interleukin-6 is associated with a lower incidence of disease-related ischemic events in patients with giant-cell arteritis: angiogenic activity of interleukin-6 as a potential protective mechanism.
Hernández-Rodríguez J, Segarra M, Vilardell C, Sánchez M, García-Martínez A, Esteban MJ, Grau JM, Urbano-Márquez A, Colomer D, Kleinman HK, Cid MC. Hernández-Rodríguez J, et al. Among authors: cid mc. Circulation. 2003 May 20;107(19):2428-34. doi: 10.1161/01.CIR.0000066907.83923.32. Epub 2003 May 12. Circulation. 2003. PMID: 12742994 Clinical Trial.
Tissue production of pro-inflammatory cytokines (IL-1beta, TNFalpha and IL-6) correlates with the intensity of the systemic inflammatory response and with corticosteroid requirements in giant-cell arteritis.
Hernández-Rodríguez J, Segarra M, Vilardell C, Sánchez M, García-Martínez A, Esteban MJ, Queralt C, Grau JM, Urbano-Márquez A, Palacín A, Colomer D, Cid MC. Hernández-Rodríguez J, et al. Among authors: cid mc. Rheumatology (Oxford). 2004 Mar;43(3):294-301. doi: 10.1093/rheumatology/keh058. Epub 2003 Dec 16. Rheumatology (Oxford). 2004. PMID: 14679293
256 results