Joner G - Search Results

1

[Genetic and immunologic risks for development of type 1 diabetes--experiences from an intervention trial].

Undlien DE, Joner G, Dahl-Jørgensen K, Rønningen KS, Nicol-Smith L, Torjesen PA, Søvik O. Undlien DE, et al. Among authors: joner g. Tidsskr Nor Laegeforen. 2000 Sep 30;120(23):2799-803. Tidsskr Nor Laegeforen. 2000. PMID: 11107929 Free article. Clinical Trial. Norwegian.

2

[Can type 1 diabetes be prevented?].

Dahl-Jørgensen K, Glosli H, Joner G, Rønningen KS, Søvik O. Dahl-Jørgensen K, et al. Among authors: joner g. Tidsskr Nor Laegeforen. 1992 Sep 30;112(23):2951-2. Tidsskr Nor Laegeforen. 1992. PMID: 1412340 Norwegian. No abstract available.

3

HLA-encoded genetic predisposition in IDDM: DR4 subtypes may be associated with different degrees of protection.

Undlien DE, Friede T, Rammensee HG, Joner G, Dahl-Jørgensen K, Søvik O, Akselsen HE, Knutsen I, Rønningen KS, Thorsby E. Undlien DE, et al. Among authors: joner g. Diabetes. 1997 Jan;46(1):143-9. doi: 10.2337/diab.46.1.143. Diabetes. 1997. PMID: 8971095

4

Polymorphisms in the cathepsin L2 (CTSL2) gene show association with type 1 diabetes and early-onset myasthenia gravis.

Viken MK, Sollid HD, Joner G, Dahl-Jørgensen K, Rønningen KS, Undlien DE, Flatø B, Selvaag AM, Førre Ø, Kvien TK, Thorsby E, Melms A, Tolosa E, Lie BA. Viken MK, et al. Among authors: joner g. Hum Immunol. 2007 Sep;68(9):748-55. doi: 10.1016/j.humimm.2007.05.009. Epub 2007 Jun 28. Hum Immunol. 2007. PMID: 17869649

5

An inverse association between history of childhood eczema and subsequent risk of type 1 diabetes that is not likely to be explained by HLA-DQ, PTPN22, or CTLA4 polymorphisms.

Stene LC, Rønningen KS, Bjørnvold M, Undlien DE, Joner G. Stene LC, et al. Among authors: joner g. Pediatr Diabetes. 2010 Sep;11(6):386-93. doi: 10.1111/j.1399-5448.2009.00605.x. Epub 2009 Nov 4. Pediatr Diabetes. 2010. PMID: 19895409

6

Does the relative risk for type 1 diabetes conferred by HLA-DQ, INS, and PTPN22 polymorphisms vary with maternal age, birth weight, or cesarean section?

Stene LC, Rønningen KS, Undlien DE, Joner G. Stene LC, et al. Among authors: joner g. Pediatr Diabetes. 2011 Mar;12(2):91-4. doi: 10.1111/j.1399-5448.2010.00669.x. Epub 2010 Sep 6. Pediatr Diabetes. 2011. PMID: 21352425

7

Joint effects of HLA, INS, PTPN22 and CTLA4 genes on the risk of type 1 diabetes.

Bjørnvold M, Undlien DE, Joner G, Dahl-Jørgensen K, Njølstad PR, Akselsen HE, Gervin K, Rønningen KS, Stene LC. Bjørnvold M, et al. Among authors: joner g. Diabetologia. 2008 Apr;51(4):589-96. doi: 10.1007/s00125-008-0932-0. Epub 2008 Feb 22. Diabetologia. 2008. PMID: 18292987 Free PMC article.

8

Genetic variants of the HLA-A, HLA-B and AIF1 loci show independent associations with type 1 diabetes in Norwegian families.

Eike MC, Olsson M, Undlien DE, Dahl-Jørgensen K, Joner G, Rønningen KS, Thorsby E, Lie BA. Eike MC, et al. Among authors: joner g. Genes Immun. 2009 Mar;10(2):141-50. doi: 10.1038/gene.2008.88. Epub 2008 Nov 6. Genes Immun. 2009. PMID: 18987644

9

[Care of children and adolescents with diabetes mellitus in Norway. A questionnaire study among diabetic patients detected over a 5-year period].

Joner G, Søvik O. Joner G, et al. Tidsskr Nor Laegeforen. 1984 May 10;104(13):852-5. Tidsskr Nor Laegeforen. 1984. PMID: 6463966 Norwegian. No abstract available.

10

No independent associations of LMP2 and LMP7 polymorphisms with susceptibility to develop IDDM.

Undlien DE, Akselsen HE, Joner G, Dahl-Jørgensen K, Søvik O, Rønningen KS, Thorsby E. Undlien DE, et al. Among authors: joner g. Diabetes. 1997 Feb;46(2):307-12. doi: 10.2337/diab.46.2.307. Diabetes. 1997. PMID: 9000709

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