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CCL19 is constitutively expressed in the CNS, up-regulated in neuroinflammation, active and also inactive multiple sclerosis lesions.
Krumbholz M, Theil D, Steinmeyer F, Cepok S, Hemmer B, Hofbauer M, Farina C, Derfuss T, Junker A, Arzberger T, Sinicina I, Hartle C, Newcombe J, Hohlfeld R, Meinl E. Krumbholz M, et al. Among authors: cepok s. J Neuroimmunol. 2007 Oct;190(1-2):72-9. doi: 10.1016/j.jneuroim.2007.07.024. Epub 2007 Sep 6. J Neuroimmunol. 2007. PMID: 17825430
Evidence for VAV2 and ZNF433 as susceptibility genes for multiple sclerosis.
Nischwitz S, Cepok S, Kroner A, Wolf C, Knop M, Müller-Sarnowski F, Pfister H, Roeske D, Rieckmann P, Hemmer B, Ising M, Uhr M, Bettecken T, Holsboer F, Müller-Myhsok B, Weber F. Nischwitz S, et al. Among authors: cepok s. J Neuroimmunol. 2010 Oct 8;227(1-2):162-6. doi: 10.1016/j.jneuroim.2010.06.003. Epub 2010 Jul 2. J Neuroimmunol. 2010. PMID: 20598377
The role of antibodies in multiple sclerosis.
Weber MS, Hemmer B, Cepok S. Weber MS, et al. Among authors: cepok s. Biochim Biophys Acta. 2011 Feb;1812(2):239-45. doi: 10.1016/j.bbadis.2010.06.009. Epub 2010 Jun 25. Biochim Biophys Acta. 2011. PMID: 20600871 Free article. Review.
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