Inflammation, Hemolysis, and Erythropoiesis Lead to Competitive Regulation of Hepcidin and Possibly Systemic Iron Status in Sickle Cell Disease
EBioMedicine
.
2018 Aug:34:8-9.
doi: 10.1016/j.ebiom.2018.07.023.
Epub 2018 Jul 31.
Authors
Yelena Z Ginzburg
1
,
Jeffrey Glassberg
2
Affiliations
1
Division of Hematology Oncology, Icahn School of Medicine at Mount Sinai, New York, NY, United States. Electronic address: yelena.ginzburg@mssm.edu.
2
Division of Hematology Oncology, Icahn School of Medicine at Mount Sinai, New York, NY, United States.
PMID:
30076048
PMCID:
PMC6116425
DOI:
10.1016/j.ebiom.2018.07.023
No abstract available
Publication types
Review
MeSH terms
Anemia, Sickle Cell / metabolism*
Anemia, Sickle Cell / pathology
Erythropoiesis*
Hemolysis*
Hepcidins / metabolism*
Humans
Inflammation / metabolism
Inflammation / pathology
Iron / metabolism*
Substances
Hepcidins
Iron
Grants and funding
R01 DK107670/DK/NIDDK NIH HHS/United States