Cardiovascular molecular mechanisms of disease with COVID-19

J Mol Cell Cardiol. 2020 Apr:141:107. doi: 10.1016/j.yjmcc.2020.04.010. Epub 2020 Apr 11.
No abstract available

MeSH terms

  • Acute Disease
  • Angiotensin-Converting Enzyme 2
  • Angiotensin-Converting Enzyme Inhibitors / therapeutic use
  • Betacoronavirus / pathogenicity*
  • COVID-19
  • Cardiovascular Diseases / diagnosis
  • Cardiovascular Diseases / epidemiology
  • Cardiovascular Diseases / etiology
  • Cardiovascular Diseases / genetics*
  • Cardiovascular System / pathology
  • Cardiovascular System / virology
  • Chronic Disease
  • Comorbidity
  • Coronavirus Infections / complications*
  • Coronavirus Infections / diagnosis
  • Coronavirus Infections / epidemiology
  • Coronavirus Infections / genetics
  • Disease Susceptibility / chemically induced
  • Host-Parasite Interactions / genetics
  • Humans
  • Hypertension / drug therapy
  • Hypertension / genetics
  • Pandemics
  • Peptidyl-Dipeptidase A / genetics
  • Peptidyl-Dipeptidase A / physiology
  • Pneumonia, Viral / complications*
  • Pneumonia, Viral / diagnosis
  • Pneumonia, Viral / epidemiology
  • Pneumonia, Viral / genetics
  • Prognosis
  • Publishing
  • Risk Factors
  • SARS-CoV-2
  • Signal Transduction / genetics

Substances

  • Angiotensin-Converting Enzyme Inhibitors
  • Peptidyl-Dipeptidase A
  • ACE2 protein, human
  • Angiotensin-Converting Enzyme 2