Bernardi P - Search Results

1

Di Lisa F, Canton M, Menabò R, Kaludercic N, Bernardi P. Di Lisa F, et al. Among authors: bernardi p. Heart Fail Rev. 2007 Dec;12(3-4):249-60. doi: 10.1007/s10741-007-9028-z. Heart Fail Rev. 2007. PMID: 17516167 Review.

2

Mitochondrial function as a determinant of recovery or death in cell response to injury.

Di Lisa F, Bernardi P. Di Lisa F, et al. Among authors: bernardi p. Mol Cell Biochem. 1998 Jul;184(1-2):379-91. Mol Cell Biochem. 1998. PMID: 9746332

3

Imaging the mitochondrial permeability transition pore in intact cells.

Petronilli V, Miotto G, Canton M, Colonna R, Bernardi P, Di Lisa F. Petronilli V, et al. Among authors: bernardi p. Biofactors. 1998;8(3-4):263-72. doi: 10.1002/biof.5520080314. Biofactors. 1998. PMID: 9914828 Review.

4

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence.

Petronilli V, Miotto G, Canton M, Brini M, Colonna R, Bernardi P, Di Lisa F. Petronilli V, et al. Among authors: bernardi p. Biophys J. 1999 Feb;76(2):725-34. doi: 10.1016/S0006-3495(99)77239-5. Biophys J. 1999. PMID: 9929477 Free PMC article.

5

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.

Scorrano L, Petronilli V, Di Lisa F, Bernardi P. Scorrano L, et al. Among authors: bernardi p. J Biol Chem. 1999 Aug 6;274(32):22581-5. doi: 10.1074/jbc.274.32.22581. J Biol Chem. 1999. PMID: 10428836 Free article.

By using a novel in situ assay based on the fluorescence changes of mitochondrially entrapped calcein (Petronilli, V., Miotto, G., Canton, M., Colonna, R., Bernardi, P., and Di Lisa, F. (1999) Biophys. J. 76, 725-734), we unequivocally show that GD3 ganglioside also …

By using a novel in situ assay based on the fluorescence changes of mitochondrially entrapped calcein (Petronilli, V., Miotto, G., Canton, M …

6

Chloromethyltetramethylrosamine (Mitotracker Orange) induces the mitochondrial permeability transition and inhibits respiratory complex I. Implications for the mechanism of cytochrome c release.

Scorrano L, Petronilli V, Colonna R, Di Lisa F, Bernardi P. Scorrano L, et al. Among authors: bernardi p. J Biol Chem. 1999 Aug 27;274(35):24657-63. doi: 10.1074/jbc.274.35.24657. J Biol Chem. 1999. PMID: 10455132 Free article.

7

Mitochondria and cell death. Mechanistic aspects and methodological issues.

Bernardi P, Scorrano L, Colonna R, Petronilli V, Di Lisa F. Bernardi P, et al. Eur J Biochem. 1999 Sep;264(3):687-701. doi: 10.1046/j.1432-1327.1999.00725.x. Eur J Biochem. 1999. PMID: 10491114 Free article. Review.

8

Interactions of chloromethyltetramethylrosamine (Mitotracker Orange) with isolated mitochondria and intact cells.

Scorrano L, Petronilli V, Colonna R, Di Lisa F, Bernardi P. Scorrano L, et al. Among authors: bernardi p. Ann N Y Acad Sci. 1999;893:391-5. doi: 10.1111/j.1749-6632.1999.tb07863.x. Ann N Y Acad Sci. 1999. PMID: 10672275 No abstract available.

9

Mitochondria in muscle cell death.

Bernardi P. Bernardi P. Ital J Neurol Sci. 1999 Dec;20(6):395-400. doi: 10.1007/s100720050057. Ital J Neurol Sci. 1999. PMID: 10937859 Review.

10

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

Di Lisa F, Menabò R, Canton M, Barile M, Bernardi P. Di Lisa F, et al. Among authors: bernardi p. J Biol Chem. 2001 Jan 26;276(4):2571-5. doi: 10.1074/jbc.M006825200. Epub 2000 Nov 9. J Biol Chem. 2001. PMID: 11073947 Free article.

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