Marcu KB - Search Results

1

Inhibitor of NF-kappa B kinases alpha and beta are both essential for high mobility group box 1-mediated chemotaxis [corrected].

Penzo M, Molteni R, Suda T, Samaniego S, Raucci A, Habiel DM, Miller F, Jiang HP, Li J, Pardi R, Palumbo R, Olivotto E, Kew RR, Bianchi ME, Marcu KB. Penzo M, et al. Among authors: marcu kb. J Immunol. 2010 Apr 15;184(8):4497-509. doi: 10.4049/jimmunol.0903131. Epub 2010 Mar 15. J Immunol. 2010. PMID: 20231695 Free PMC article.

2

The IKKα-dependent NF-κB p52/RelB noncanonical pathway is essential to sustain a CXCL12 autocrine loop in cells migrating in response to HMGB1.

Kew RR, Penzo M, Habiel DM, Marcu KB. Kew RR, et al. Among authors: marcu kb. J Immunol. 2012 Mar 1;188(5):2380-6. doi: 10.4049/jimmunol.1102454. Epub 2012 Jan 27. J Immunol. 2012. PMID: 22287708 Free PMC article.

3

Sustained NF-kappaB activation produces a short-term cell proliferation block in conjunction with repressing effectors of cell cycle progression controlled by E2F or FoxM1.

Penzo M, Massa PE, Olivotto E, Bianchi F, Borzi RM, Hanidu A, Li X, Li J, Marcu KB. Penzo M, et al. Among authors: marcu kb. J Cell Physiol. 2009 Jan;218(1):215-27. doi: 10.1002/jcp.21596. J Cell Physiol. 2009. PMID: 18803232 Free PMC article.

4

Cell migration to CXCL12 requires simultaneous IKKα and IKKβ-dependent NF-κB signaling.

Penzo M, Habiel DM, Ramadass M, Kew RR, Marcu KB. Penzo M, et al. Among authors: marcu kb. Biochim Biophys Acta. 2014 Sep;1843(9):1796-1804. doi: 10.1016/j.bbamcr.2014.04.011. Epub 2014 Apr 18. Biochim Biophys Acta. 2014. PMID: 24747690 Free PMC article.

5

Cells migrating to sites of tissue damage in response to the danger signal HMGB1 require NF-kappaB activation.

Palumbo R, Galvez BG, Pusterla T, De Marchis F, Cossu G, Marcu KB, Bianchi ME. Palumbo R, et al. Among authors: marcu kb. J Cell Biol. 2007 Oct 8;179(1):33-40. doi: 10.1083/jcb.200704015. J Cell Biol. 2007. PMID: 17923528 Free PMC article.

6

Differential requirements for IKKalpha and IKKbeta in the differentiation of primary human osteoarthritic chondrocytes.

Olivotto E, Borzi RM, Vitellozzi R, Pagani S, Facchini A, Battistelli M, Penzo M, Li X, Flamigni F, Li J, Falcieri E, Facchini A, Marcu KB. Olivotto E, et al. Among authors: marcu kb. Arthritis Rheum. 2008 Jan;58(1):227-39. doi: 10.1002/art.23211. Arthritis Rheum. 2008. PMID: 18163512 Free PMC article.

7

IKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.

Samaniego S, Marcu KB. Samaniego S, et al. Among authors: marcu kb. PLoS One. 2013;8(1):e54124. doi: 10.1371/journal.pone.0054124. Epub 2013 Jan 22. PLoS One. 2013. PMID: 23349802 Free PMC article.

8

Basal and IL-1β enhanced chondrocyte chemotactic activity on monocytes are co-dependent on both IKKα and IKKβ NF-κB activating kinases.

Olivotto E, Minguzzi M, D'Adamo S, Astolfi A, Santi S, Uguccioni M, Marcu KB, Borzì RM. Olivotto E, et al. Among authors: marcu kb. Sci Rep. 2021 Nov 4;11(1):21697. doi: 10.1038/s41598-021-01063-2. Sci Rep. 2021. PMID: 34737366 Free PMC article.

9

NF-kappaB signaling: multiple angles to target OA.

Marcu KB, Otero M, Olivotto E, Borzi RM, Goldring MB. Marcu KB, et al. Curr Drug Targets. 2010 May;11(5):599-613. doi: 10.2174/138945010791011938. Curr Drug Targets. 2010. PMID: 20199390 Free PMC article. Review.

10

Pathophysiology of osteoarthritis: canonical NF-κB/IKKβ-dependent and kinase-independent effects of IKKα in cartilage degradation and chondrocyte differentiation.

Olivotto E, Otero M, Marcu KB, Goldring MB. Olivotto E, et al. Among authors: marcu kb. RMD Open. 2015 Aug 15;1(Suppl 1):e000061. doi: 10.1136/rmdopen-2015-000061. eCollection 2015. RMD Open. 2015. PMID: 26557379 Free PMC article. Review.

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