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Lymphatic dysfunction, not aplasia, underlies Milroy disease.
Mellor RH, Hubert CE, Stanton AW, Tate N, Akhras V, Smith A, Burnand KG, Jeffery S, Mäkinen T, Levick JR, Mortimer PS. Mellor RH, et al. Among authors: smith a. Microcirculation. 2010 May;17(4):281-96. doi: 10.1111/j.1549-8719.2010.00030.x. Microcirculation. 2010. PMID: 20536741
Encapsulation of angiogenic monocytes using bio-spraying technology.
Patel AS, Smith A, Attia RQ, Mattock K, Humphries J, Lyons O, Saha P, Modarai B, Jayasinghe SN. Patel AS, et al. Among authors: smith a. Integr Biol (Camb). 2012 Jun;4(6):628-32. doi: 10.1039/c2ib20033c. Epub 2012 May 15. Integr Biol (Camb). 2012. PMID: 22588229
TIE2-expressing monocytes/macrophages regulate revascularization of the ischemic limb.
Patel AS, Smith A, Nucera S, Biziato D, Saha P, Attia RQ, Humphries J, Mattock K, Grover SP, Lyons OT, Guidotti LG, Siow R, Ivetic A, Egginton S, Waltham M, Naldini L, De Palma M, Modarai B. Patel AS, et al. Among authors: smith a. EMBO Mol Med. 2013 Jun;5(6):858-69. doi: 10.1002/emmm.201302752. Epub 2013 May 7. EMBO Mol Med. 2013. PMID: 23653322 Free PMC article.
Magnetic resonance T1 relaxation time of venous thrombus is determined by iron processing and predicts susceptibility to lysis.
Saha P, Andia ME, Modarai B, Blume U, Humphries J, Patel AS, Phinikaridou A, Evans CE, Mattock K, Grover SP, Ahmad A, Lyons OT, Attia RQ, Renné T, Premaratne S, Wiethoff AJ, Botnar RM, Schaeffter T, Waltham M, Smith A. Saha P, et al. Among authors: smith a. Circulation. 2013 Aug 13;128(7):729-736. doi: 10.1161/CIRCULATIONAHA.113.001371. Epub 2013 Jul 2. Circulation. 2013. PMID: 23820077 Free PMC article.
Antiangiogenic therapy inhibits venous thrombus resolution.
Evans CE, Grover SP, Humphries J, Saha P, Patel AP, Patel AS, Lyons OT, Waltham M, Modarai B, Smith A. Evans CE, et al. Among authors: smith a. Arterioscler Thromb Vasc Biol. 2014 Mar;34(3):565-70. doi: 10.1161/ATVBAHA.113.302998. Epub 2014 Jan 16. Arterioscler Thromb Vasc Biol. 2014. PMID: 24436367
Human venous valve disease caused by mutations in FOXC2 and GJC2.
Lyons O, Saha P, Seet C, Kuchta A, Arnold A, Grover S, Rashbrook V, Sabine A, Vizcay-Barrena G, Patel A, Ludwinski F, Padayachee S, Kume T, Kwak BR, Brice G, Mansour S, Ostergaard P, Mortimer P, Jeffery S, Brown N, Makinen T, Petrova TV, Modarai B, Smith A. Lyons O, et al. Among authors: smith a. J Exp Med. 2017 Aug 7;214(8):2437-2452. doi: 10.1084/jem.20160875. J Exp Med. 2017. PMID: 28724617 Free PMC article.
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