Havasi V - Search Results

1

RNA binding proteins PTBP1 and HNRNPL regulate CFTR mRNA decay.

Siddiqui A, Saxena A, Echols J, Havasi V, Fu L, Keeling KM. Siddiqui A, et al. Among authors: havasi v. Heliyon. 2023 Nov 13;9(11):e22281. doi: 10.1016/j.heliyon.2023.e22281. eCollection 2023 Nov. Heliyon. 2023. PMID: 38045134 Free PMC article.

2

A regulated NMD mouse model supports NMD inhibition as a viable therapeutic option to treat genetic diseases.

Echols J, Siddiqui A, Dai Y, Havasi V, Sun R, Kaczmarczyk A, Keeling KM. Echols J, et al. Among authors: havasi v. Dis Model Mech. 2020 Aug 27;13(8):dmm044891. doi: 10.1242/dmm.044891. Dis Model Mech. 2020. PMID: 32737261 Free PMC article.

3

Reduced bone length, growth plate thickness, bone content, and IGF-I as a model for poor growth in the CFTR-deficient rat.

Stalvey MS, Havasi V, Tuggle KL, Wang D, Birket S, Rowe SM, Sorscher EJ. Stalvey MS, et al. Among authors: havasi v. PLoS One. 2017 Nov 30;12(11):e0188497. doi: 10.1371/journal.pone.0188497. eCollection 2017. PLoS One. 2017. PMID: 29190650 Free PMC article.

4

Increasing the Endoplasmic Reticulum Pool of the F508del Allele of the Cystic Fibrosis Transmembrane Conductance Regulator Leads to Greater Folding Correction by Small Molecule Therapeutics.

Chung WJ, Goeckeler-Fried JL, Havasi V, Chiang A, Rowe SM, Plyler ZE, Hong JS, Mazur M, Piazza GA, Keeton AB, White EL, Rasmussen L, Weissman AM, Denny RA, Brodsky JL, Sorscher EJ. Chung WJ, et al. Among authors: havasi v. PLoS One. 2016 Oct 12;11(10):e0163615. doi: 10.1371/journal.pone.0163615. eCollection 2016. PLoS One. 2016. PMID: 27732613 Free PMC article.

5

Re: Lu et al.: Different cystic fibrosis transmembrane conductance regulator mutations in Chinese men with congenital bilateral absence of the vas deferens and other acquired obstructive azoospermia (Urology 2013;82:824-828).

Havasi V. Havasi V. Urology. 2014 Mar;83(3):678-9. doi: 10.1016/j.urology.2013.11.026. Urology. 2014. PMID: 24581531 No abstract available.

6

Osteoblast CFTR inactivation reduces differentiation and osteoprotegerin expression in a mouse model of cystic fibrosis-related bone disease.

Stalvey MS, Clines KL, Havasi V, McKibbin CR, Dunn LK, Chung WJ, Clines GA. Stalvey MS, et al. Among authors: havasi v. PLoS One. 2013 Nov 13;8(11):e80098. doi: 10.1371/journal.pone.0080098. eCollection 2013. PLoS One. 2013. PMID: 24236172 Free PMC article.

7

The silent codon change I507-ATC->ATT contributes to the severity of the ΔF508 CFTR channel dysfunction.

Lazrak A, Fu L, Bali V, Bartoszewski R, Rab A, Havasi V, Keiles S, Kappes J, Kumar R, Lefkowitz E, Sorscher EJ, Matalon S, Collawn JF, Bebok Z. Lazrak A, et al. Among authors: havasi v. FASEB J. 2013 Nov;27(11):4630-45. doi: 10.1096/fj.13-227330. Epub 2013 Aug 1. FASEB J. 2013. PMID: 23907436 Free PMC article.

8

Association of cystic fibrosis genetic modifiers with congenital bilateral absence of the vas deferens.

Havasi V, Rowe SM, Kolettis PN, Dayangac D, Sahin A, Grangeia A, Carvalho F, Barros A, Sousa M, Bassas L, Casals T, Sorscher EJ. Havasi V, et al. Fertil Steril. 2010 Nov;94(6):2122-7. doi: 10.1016/j.fertnstert.2009.11.044. Epub 2010 Jan 25. Fertil Steril. 2010. PMID: 20100616 Free PMC article.

9

Trends in the epidemiology of human G1P[8] rotaviruses: a hungarian study.

Bányai K, Gentsch JR, Martella V, Bogdán A, Havasi V, Kisfali P, Szabó A, Mihály I, Molnár P, Melegh B, Szücs G. Bányai K, et al. Among authors: havasi v. J Infect Dis. 2009 Nov 1;200 Suppl 1:S222-7. doi: 10.1086/605052. J Infect Dis. 2009. PMID: 19821713

10

The role of the F508C mutation in congenital bilateral absence of the vas deferens.

Havasi V, Keiles S, Hambuch T, Sorscher EJ, Kammesheidt A. Havasi V, et al. Genet Med. 2008 Dec;10(12):910-4. doi: 10.1097/GIM.0b013e31818e594d. Genet Med. 2008. PMID: 19092444 Free article.

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