Nakamura M - Search Results

1

Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor protein.

Smith DH, Nakamura M, McIntosh TK, Wang J, Rodríguez A, Chen XH, Raghupathi R, Saatman KE, Clemens J, Schmidt ML, Lee VM, Trojanowski JQ. Smith DH, et al. Among authors: nakamura m. Am J Pathol. 1998 Sep;153(3):1005-10. doi: 10.1016/s0002-9440(10)65643-x. Am J Pathol. 1998. PMID: 9736050 Free PMC article.

2

Differential acute and chronic responses of tumor necrosis factor-deficient mice to experimental brain injury.

Scherbel U, Raghupathi R, Nakamura M, Saatman KE, Trojanowski JQ, Neugebauer E, Marino MW, McIntosh TK. Scherbel U, et al. Among authors: nakamura m. Proc Natl Acad Sci U S A. 1999 Jul 20;96(15):8721-6. doi: 10.1073/pnas.96.15.8721. Proc Natl Acad Sci U S A. 1999. PMID: 10411942 Free PMC article.

3

Increased vulnerability of NFH-LacZ transgenic mouse to traumatic brain injury-induced behavioral deficits and cortical damage.

Nakamura M, Saatman KE, Galvin JE, Scherbel U, Raghupathi R, Trojanowski JQ, McIntosh TK. Nakamura M, et al. J Cereb Blood Flow Metab. 1999 Jul;19(7):762-70. doi: 10.1097/00004647-199907000-00006. J Cereb Blood Flow Metab. 1999. PMID: 10413031

4

Traumatic brain injury in young, amyloid-beta peptide overexpressing transgenic mice induces marked ipsilateral hippocampal atrophy and diminished Abeta deposition during aging.

Nakagawa Y, Nakamura M, McIntosh TK, Rodriguez A, Berlin JA, Smith DH, Saatman KE, Raghupathi R, Clemens J, Saido TC, Schmidt ML, Lee VM, Trojanowski JQ. Nakagawa Y, et al. Among authors: nakamura m. J Comp Neurol. 1999 Aug 30;411(3):390-8. J Comp Neurol. 1999. PMID: 10413774

5

Overexpression of Bcl-2 is neuroprotective after experimental brain injury in transgenic mice.

Nakamura M, Raghupathi R, Merry DE, Scherbel U, Saatman KE, Mcintosh TK. Nakamura M, et al. J Comp Neurol. 1999 Oct 4;412(4):681-92. doi: 10.1002/(sici)1096-9861(19991004)412:4<681::aid-cne9>3.0.co;2-f. J Comp Neurol. 1999. PMID: 10464363

6

Brain trauma in aged transgenic mice induces regression of established abeta deposits.

Nakagawa Y, Reed L, Nakamura M, McIntosh TK, Smith DH, Saatman KE, Raghupathi R, Clemens J, Saido TC, Lee VM, Trojanowski JQ. Nakagawa Y, et al. Among authors: nakamura m. Exp Neurol. 2000 May;163(1):244-52. doi: 10.1006/exnr.2000.7375. Exp Neurol. 2000. PMID: 10785464

7

Neurofilament-rich intraneuronal inclusions exacerbate neurodegenerative sequelae of brain trauma in NFH/LacZ transgenic mice.

Galvin JE, Nakamura M, McIntosh TK, Saatman KE, Sampathu D, Raghupathi R, Lee VM, Trojanowski JQ. Galvin JE, et al. Among authors: nakamura m. Exp Neurol. 2000 Sep;165(1):77-89. doi: 10.1006/exnr.2000.7461. Exp Neurol. 2000. PMID: 10964487 Free article.

8

Age-dependent synuclein pathology following traumatic brain injury in mice.

Uryu K, Giasson BI, Longhi L, Martinez D, Murray I, Conte V, Nakamura M, Saatman K, Talbot K, Horiguchi T, McIntosh T, Lee VM, Trojanowski JQ. Uryu K, et al. Among authors: nakamura m. Exp Neurol. 2003 Nov;184(1):214-24. doi: 10.1016/s0014-4886(03)00245-0. Exp Neurol. 2003. PMID: 14637093 Free article.

9

The loss of Cbl-phosphatidylinositol 3-kinase interaction perturbs RANKL-mediated signaling, inhibiting bone resorption and promoting osteoclast survival.

Adapala NS, Barbe MF, Langdon WY, Nakamura MC, Tsygankov AY, Sanjay A. Adapala NS, et al. J Biol Chem. 2010 Nov 19;285(47):36745-58. doi: 10.1074/jbc.M110.124628. Epub 2010 Sep 17. J Biol Chem. 2010. PMID: 20851882 Free PMC article.

10

Identification of the hybrid gene LILRB5-3 by long-read sequencing and implication of its novel signaling function.

Hirayasu K, Khor SS, Kawai Y, Shimada M, Omae Y, Hasegawa G, Hashikawa Y, Tanimoto H, Ohashi J, Hosomichi K, Tajima A, Nakamura H, Nakamura M, Tokunaga K, Hanayama R, Nagasaki M. Hirayasu K, et al. Among authors: nakamura m. Front Immunol. 2024 May 14;15:1398935. doi: 10.3389/fimmu.2024.1398935. eCollection 2024. Front Immunol. 2024. PMID: 38807600 Free PMC article.

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