Napolitano C - Search Results

1

Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism.

Splawski I, Timothy KW, Sharpe LM, Decher N, Kumar P, Bloise R, Napolitano C, Schwartz PJ, Joseph RM, Condouris K, Tager-Flusberg H, Priori SG, Sanguinetti MC, Keating MT. Splawski I, et al. Among authors: napolitano c. Cell. 2004 Oct 1;119(1):19-31. doi: 10.1016/j.cell.2004.09.011. Cell. 2004. PMID: 15454078 Free article.

2

Electrophysiologic mechanisms involved in the development of torsades de pointes.

Priori SG, Napolitano C, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Cardiovasc Drugs Ther. 1991 Feb;5(1):203-12. doi: 10.1007/BF03029822. Cardiovasc Drugs Ther. 1991. PMID: 2036341 Review.

3

Early afterdepolarizations induced in vivo by reperfusion of ischemic myocardium. A possible mechanism for reperfusion arrhythmias.

Priori SG, Mantica M, Napolitano C, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Circulation. 1990 Jun;81(6):1911-20. doi: 10.1161/01.cir.81.6.1911. Circulation. 1990. PMID: 2344683

4

Torsade de pointes. Mechanisms and management.

Napolitano C, Priori SG, Schwartz PJ. Napolitano C, et al. Drugs. 1994 Jan;47(1):51-65. doi: 10.2165/00003495-199447010-00004. Drugs. 1994. PMID: 7510621 Review.

5

Sympathetic activation, ventricular repolarization and Ikr blockade: implications for the antifibrillatory efficacy of potassium channel blocking agents.

Vanoli E, Priori SG, Nakagawa H, Hirao K, Napolitano C, Diehl L, Lazzara R, Schwartz PJ. Vanoli E, et al. Among authors: napolitano c. J Am Coll Cardiol. 1995 Jun;25(7):1609-14. doi: 10.1016/0735-1097(95)00046-7. J Am Coll Cardiol. 1995. PMID: 7759713 Free article.

6

Dispersion of the QT interval. A marker of therapeutic efficacy in the idiopathic long QT syndrome.

Priori SG, Napolitano C, Diehl L, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Circulation. 1994 Apr;89(4):1681-9. doi: 10.1161/01.cir.89.4.1681. Circulation. 1994. PMID: 7908611

7

Cardiac receptor activation and arrhythmogenesis.

Priori SG, Napolitano C, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Eur Heart J. 1993 Sep;14 Suppl E:20-6. doi: 10.1093/eurheartj/14.suppl_e.20. Eur Heart J. 1993. PMID: 8223751 Review.

8

Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy.

Schwartz PJ, Priori SG, Locati EH, Napolitano C, Cantù F, Towbin JA, Keating MT, Hammoude H, Brown AM, Chen LS, Colatsky TJ. Schwartz PJ, et al. Among authors: napolitano c. Circulation. 1995 Dec 15;92(12):3381-6. doi: 10.1161/01.cir.92.12.3381. Circulation. 1995. PMID: 8521555

9

Differential response to Na+ channel blockade, beta-adrenergic stimulation, and rapid pacing in a cellular model mimicking the SCN5A and HERG defects present in the long-QT syndrome.

Priori SG, Napolitano C, Cantù F, Brown AM, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Circ Res. 1996 Jun;78(6):1009-15. doi: 10.1161/01.res.78.6.1009. Circ Res. 1996. PMID: 8635231

10

A molecular basis for the therapy of the long QT syndrome.

Priori SG, Napolitano C, Schwartz PJ. Priori SG, et al. Among authors: napolitano c. Arch Mal Coeur Vaiss. 1996 Sep;89(9):1185-7. Arch Mal Coeur Vaiss. 1996. PMID: 8952843

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